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A new study by scientists at The Scripps Research Institute (TSRI) shows that the natural acne-fighting drug deca (known as BHT) is not only safe for acne but it also works in a way that helps the body repair damaged acne.
Deca is one of the few natural anti-acne drugs that works by acting as a sort of natural skin barrier.
The TSHR researchers used a combination of genetic manipulation, skin test, and other lab studies to find that deca does not work like other natural anti-(meth-)acne medications.
The findings have been published online in the journal Science Translational Medicine.
The researchers were interested in finding out whether deca could act as a natural skin-repair agent because, at least in humans, skin repair is the primary function of these medications.
They tested two different ways of removing the deca from a mouse model of acne.
The first is to inject the drug into the skin and let the drug penetrate the skin in a small amount.
The second is to use a synthetic version of the drug.
The drug did not affect the ability of the mouse skin to repair damaged skin cells.
However, it did have a very strong effect on the skin cells, which were able to repair more quickly and to recover more quickly than normal skin.
In other words, deca did not just act like a skin-damaging drug.
It was able to improve the healing process of damaged skin.
The results also indicated that decas ability to repair acne caused an increase in the production of new skin cells that can help repair damaged areas of the skin.
Decaprolin has also been shown to help in the treatment of acne by reducing inflammation and the production and release of scar tissue.
In a separate study, researchers at TSSI used a different kind of synthetic version (dexacycline) of deca.
When the researchers injected the synthetic version into the same mice, they saw the drug acting in the same way as the natural deca by promoting a production of scar cells.
When they injected deca into a group of mice with healthy skin, the synthetic drug reduced the amount of scar cell production.
The deca treatment also had the same effect as the synthetic deca when it was given to the mice with damaged skin, suggesting that it could act like an anti-inflammatory drug.
These findings are very encouraging.
Decapping a drug to have a similar effect to natural decaprolins results in more than a 50 percent decrease in the amount that scar cells produce.
The study shows that this natural anti-[meth]-acne drug is an effective way to help restore damaged skin and the skin cell regeneration process.
The next step in the research is to do a better test in human patients to see if there is a way to do this with an injectable deca drug that works similarly to the synthetic one.
The research is supported by the National Institutes of Health (grants R01 HD 025793 and R01 DK-082638) and the National Science Foundation.
More about deca: A synthetic drug that reduces the production & release of new acne-causing skin cells Researchers at the Scrippes Research Institute have found that decaprolerine is not just a skin repair drug, but also has a positive effect on human skin.
Using a synthetic decaprostane analogue, they found that the synthetic dose produced more new scar cells than the natural dose did.
This was shown by using the same assay to measure the new scar cell count in skin samples from patients with mild acne.
A synthetic decaperine analogue, dexacycroline, also produced scar cells more rapidly than the synthetic dosage did.
The authors suggest that the difference in scar cell formation between the synthetic and the natural doses could be due to the fact that the compound has a strong antimicrobial activity.
In addition, the researchers found that when decaproprolerines active ingredients are combined with topical retinoids, they also stimulate scar cell development.
A recent study by TSRI researchers also looked at the use of synthetic decapsone, a synthetic form of decaprone, in the prevention of scarring and wound healing.
In that study, they used an oral decaprocolline-containing product, decaprone-3-yl, as a treatment to prevent scarring, wound healing, and inflammation.
The TSRI team found that a combination treatment of decapsoone with a topical retinoic acid-based retinoid cream reduced the formation of new scar tissue in the skin, increased collagen production, and restored wound healing in patients with moderate to severe acne.
This is very exciting news, and a big step forward for the future of anti-Acne drugs.
The new research also